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results (Fairburn et al, 1999) The heritability of AN was estimated to be 76% in another population-based twin sample (Klump et al, 2001) and 58% in a bivariate analysis of AN and major depression (Wade et al, 2000) The magnitude of the genetic contribution to AN remains unresolved Large-scale twin studies are needed to de ne the extent and nature of the genetic and environmental contributions to the aetiology of AN Three clinically ascertained twin pair studies of BN (Fichter & Noegal, 1990; Hsu et al, 1990; Treasure & Holland, 1989) and two population-based twin studies of BN (Wade et al, 1999; Bulik et al, 1998; Kendler et al, 1991, 1995) have been conducted and have consistently demonstrated signi cant genetic contributions in the liability to BN.

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Reanalysis of the data from the twin studies of BN produced estimates of heritabilty ranging from 31% to 83% (Bulik et al, 2000) In general non-shared environmental effects were shown to account for the remaining variance in liability to BN The magnitude of the contribution of shared environmental effects is unclear but in the majority of the twin studies it appears to be less important than additive genetic effects and non-shared environmental effects (Bulik et al, 2000) Several of the symptoms, behaviours and attitudes associated with disordered eating have been shown to be heritable in different populations of twins These continuous traits are assessed using psychometric questionnaires, such as the Eating Disorder Inventory (EDI; Garner et al, 1984) and the Eating Disorders Examination (EDE; Fairburn & Cooper, 1993).

This is so because in most cases a single key is used to encrypt many blocks before it is changed and thus KSA can spend more time on randomizing things than on the encryption function Due to this reasoning, in many cases, the analysis of key scheduling function is hard It is also hardly worth the effort since in most cases the awed key schedule can be replaced without altering the main encryption function An attacker may assume that subkeys are independent random variables If the cipher is broken under this assumption, no patch of key schedule will save it Interestingly, it is possible to avoid the need for a complex key schedule by using a xed mixing permutation on a large set of inputs and two keys XORed at the input and at the output of the encryption function [17,34] These keys are now called whitening keys.

The Drive for Thinness subscale of the EDI, was shown to be heritable in one twin population (Holland et al, 1988), and in another twin study several EDI subscales showed heritabilities ranging from 28% to 52% (Rutherford et al, 1993) Heritabilities of 46% and 70% have been reported for binge eating and self-induced vomiting respectively (Sullivan et al, 1998) There is evidence of age-related differences in genetic and environmental in uences on these traits Marked differences in heritabilities for EDI subscales have been reported for a preadolescent (aged 11 years) and an adolescent (aged 17 years) twin sample from the same population (Klump et al, 2000) The contribution of additive genetic effects for the EDI subscales was greater in the adolescent twin sample than in the preadolescent group.

terms are clari ed as far as possible But second, it must not be thought that, merely by clarifying the meanings of words, real problems in the real world can be solved The clari cation of meaning is an essential part of inquiry into ethics, but it is only a part Good For example, in order to work out what it is good to do it is important to stress that the word good has at least three separate meanings a Good as a description of objects that are useful People talk of a good clock or a good motor car using this sense of good .

Based on this nding it has been proposed that puberty may activate the heritability of eating disorders (Klump et al, 2001) Measures from the EDE such as dietary restraint, and concerns about eating, weight and shape also appear to be heritable (Wade et al, 1998) Several family and twin studies have investigated the causes of comorbidity between eating disorders and personality traits and other psychiatric disorders Family studies investigating the relationship between personality traits and eating disorders have shown that some personality traits are signi cantly elevated in the unaffected relatives of probands with an eating disorder compared to the relatives of the control group (Kaye et al, 1999; Lilenfeld et al, 2000) For example perfectionism, ineffectiveness, and interpersonal distrust has been found to be signi cantly elevated in the unaffected relatives of BN probands compared to the relatives of the control group (Lilenfeld et al.